When you’re on a record-breaking world tour, you have to keep performing even if you feel like you could get a cold. Taylor Swift showed her professionalism over the weekend as she handled a runny nose during her performance in Edinburgh.
Taylor Swift’s Unstoppable Performance in the Cold
The Pennsylvania-born pop singer was seen blowing into her fingers several times during the Scottish event before smearing it over her sparkling frock. This occurred on the first leg of her UK tour. The 34-year-old Taylor Swift’s three performances at Murrayfield Stadium last weekend drew almost 219,000 spectators.
The frigid air in the stadium did not help the singer’s plight, even though summer is just a few days away. Particularly when the pop star was seen wiping her nose while playing the smash song “Don’t Blame Me,” some have blamed the incident on the cold. The temperature fell below 46.4 degrees Fahrenheit on the night of the show. In the video, the singer can be seen pumping snot into her palms, letting it drip, and then wiping it on her skirt.
Different Reactions from the Viewers
Some fans have even accused the musical sensation of flicking mucus in their way while she braved the conditions that turned her nose red. Many on social media expressed their disgust, despite the fact that several people offered their sympathies. Someone said, “Taylor Swift blew her nose and snot on her skirt on stage.” Additionally, fans have seen Taylor licking the snot out of her nose.
Another person looked horrified and yelled, “Someone please give Taylor Swift some tissues.” I’ve observed her clean her nose with her hands so many times in the previous few days that it’s absurd. I get that she’s performing, but there has to be a method to make sure she always has Kleenex.
Help from a Select Group of Fans
However, other people backed Taylor Swift with the remark, “I’m not even a Swiftie, but I seriously don’t get why anyone would be mad or disgusted by someone wiping their nose.” Really, people? What was she supposed to do? Even if it’s unlikely that she had a tissue close by, at least she wasn’t wanting snot all over her face.
The UK Tour Continues
Taylor Swift gave three performances in Edinburgh to kick off the British singer’s tour. Eight nights at Wembley and evenings in Cardiff and Liverpool will follow. where 720,000 fans will be present alone in London. Following the European tour, Taylor will make nine more stops in the United States, including Miami, New Orleans, and Indianapolis. 53 US dates have already been completed by her. She has committed to performing in fifty cities across Europe.
Fan Loyalty and Its Impact on the Economy
Several ardent Taylor Swift admirers began to create lines 48 hours before the celebrity even made it to the city for her tour. A whopping 220,000 individuals are waiting impatiently to see and buy things. However, a lot of individuals in Edinburgh had trouble getting housing due to the quick reservations made for hotels. Despite charging three times as much for the superstar’s three-night stay from June 7 to June 9.
Her Eras tour is predicted to boost the British economy by about £1 billion. Apart from the ticket price, supporters are required to cover the expenses for accommodation, travel, a pre-concert meal, and official merchandise. Many will also buy new clothes specifically for this historic occasion. According to research, each Swiftie who sees their idol perform live in the UK will pay, on average, £848.
Ticket Controversy: Excessive Demand Worldwide
Since her Scottish premiere this past weekend, UK fans have been grumbling about how tough it is to get tickets. This is a result of Americans buying UK television series. Fans expressed their dissatisfaction with some of the guests’ flights on social media after the performance, given that the artist hasn’t performed in Scotland in eight years.
Americans countered that they couldn’t get tickets in their state and remarked that dates in Europe are far less expensive because of the strong currency. 4.35 million tickets have been sold to Swiftie fans worldwide, but due to the extremely high demand, fans have had difficulty obtaining tickets to shows.
We’ve All Waited for It: Scientists Created a Drug That Can Help Regrow Lost Teeth
The tooth fairy is a welcome guest for any child who has lost a tooth. Not only will the fairy leave a small gift under the child’s pillow, but they be assured of a replacement tooth in a few months. Unfortunately, the scenario is quite different for adults grappling with a loss of teeth. Luckily, there may be some hope thanks to a new study performed by scientists at Kyoto University and the University of Fukui.
A dental breakthrough
While the typical adult mouth houses 32 teeth, approximately 1% of the population exhibits variations of them, either possessing more or fewer teeth due to congenital conditions. Researchers have delved into the genetic factors behind cases of excessive teeth, seeking valuable insights into the potential regeneration of teeth in adults. This study is the first to show that monoclonal antibodies can help regrow teeth. It suggests a new way to treat a dental problem that currently requires implants and other artificial solutions.
A bit of science
The research team disclosed that an antibody targeting a specific gene, known as uterine sensitization-associated gene-1 (USAG-1), can induce tooth development in mice affected by tooth agenesis, a congenital condition. The findings were published in the journal, Science Advances.
As per Katsu Takahashi, a senior lecturer at the Kyoto University Graduate School of Medicine and one of the principal contributors to the study, the essential molecules crucial for the development of teeth have already been pinpointed. “The morphogenesis of individual teeth depends on the interactions of several molecules including BMP, or bone morphogenetic protein, and Wnt signaling,” says Takahashi.
On April 13, 2021, the University of Kyoto posted its first pic of newly-grown teeth in mice.
BMP and Wnt are involved in more than just tooth development; they affect the growth of organs and tissues early in the body’s development. Because drugs affecting them directly might have broad side effects, scientists are cautious. To find a potentially safer method, researchers focused on the gene USAG-1, thinking that aiming at factors countering BMP and Wnt specifically in tooth development could be more precise.
“We knew that suppressing USAG-1 benefits tooth growth. What we did not know was whether it would be enough,” added Takahashi.
The first results
Scientists looked at how different monoclonal antibodies affect USAG-1. Monoclonal antibodies are often used to treat things like cancer and arthritis and for making vaccines. Tests with this antibody showed that BMP signaling is crucial for deciding the number of teeth in mice. Also, just one treatment was enough to grow a whole tooth. Further tests confirmed these positive results in ferrets too.
“Ferrets are diphyodont animals with similar dental patterns to humans. Our next plan is to test the antibodies on other animals, such as pigs and dogs,” explained Takahashi.
Fully regrown frontal teeth in ferrets
The next steps
Now, scientists are going to test the drug on healthy adults. If that goes well, the team plans to try it on kids aged 2 to 6 with a rare tooth problem called anodontia, a genetic disorder defined as the absence of all teeth. These kids will get one shot of the drug to see if it makes their teeth grow. If everything works out, the medicine might be approved by 2030.
Takahashi sees the new medicine as an additional choice for individuals who are missing some or all of their teeth.
“The idea of growing new teeth is every dentist’s dream,” Takahashi told the Japanese newspaper, The Mainichi in June this year. “I’ve been working on this since I was a graduate student. I was confident I’d be able to make it happen.”
So hopefully, by the year 2030, humans will get a chance to have their third generation of teeth grown and say goodbye to implants. Until then, make sure to keep your teeth strong and healthy — this article will help you with that.
Preview photo credit KyotoU_News / Twitter
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